What is SunPatch
The SunPatch delivers UV protection technology in a film application.
The SunPatch is virtually invisible when worn, flexible, easy to apply and remove. The SunPatch forms a comfortable ‘second skin’ with the added benefit of protection to be used all year round.SunPatch is to be used in conjunction with all other recommended sun care procedures. Avoid use on irritated skin. Should any of the following occur: Burning, Itching, Stinging of the skin; early appearance of Redness or Drying or Tightening of the skin – Please seek Medical Advice.
The Skin Epidermis
The epidermis is the outer layer of skin. The thickness of the epidermis varies in different types of skin. It is the thinnest on the eyelids at .05 mm and the thickest on the palms and soles at 1.5 mm.
The dermis also varies in thickness depending on the location of the skin. It is .3 mm on the eyelid and 3.0 mm on the back.
Subcutaneous TissueThe subcutaneous tissue is a layer of fat and connective tissue that houses larger blood vessels and nerves.This layer is important for the regulation of temperature of the skin itself and the body. The size of this layer varies throughout the body and from person to person.the-skin-images
Cause – UV light dilates the tiny veins within the skin so they show up on the surface. Also called spider veins or broken veins.
Cause – Melanomas In the UK, 12,818 people were diagnosed with these in 2010, according to Cancer Research UK. Most skin cancers are strongly linked to intermittent exposure to high-intensity sunlight. Malignant tumours that start life in the pigment-producing melanocyte cells or moles develop when DNA damage to skin cells, caused by UVA/B exposure, leads to genetic mutations that grow into a tumour. So if you have pigmentation/age spots it’s worth having them checked. There is a genetic element to melanoma, so if it runs in your family, be vigilant.
Non-melanomaCause – Skin cancer Around 100,000 people were diagnosed with this in 2010. Exposure to ultraviolet light can eventually lead to actinic keratoses, a kind of ‘pre cancer’ which if not spotted and treated may develop into basal cell carcinoma or squamous cell carcinoma. Non-melanomas are much less likely to be fatal than melanomas.
Cause – Exposure to UV light increases the number of enzymes in the skin that break down the collagen fibres that give the skin its structure.
Sagging skinCause – Along with the damage to collagen, UVA and UVB exposure slowly wrecks the skin’s elastin, a protein that gives skin its bounce.
Age spotsCause – Too much UVA and UVB exposure can damage the DNA of the melanocytes, the cells within the skin that produce the brown pigment called melanin. This makes the melanocytes enlarge and cluster together into the visible ‘liver spots’ or ‘age spots’ that often appear first on the backs of hands and the decolletage.
Rough and Dry SkinCause – The damage done by UVA/Blight to skin’s collagen and elastin harms its texture, too. As well as feeling rough to the touch, its surface doesn’t reflect light like smoother skin, so it looks dull and water escapes more easily from its cells, making it dryer.‘Groups who spend a lot of time in the sun and so are at increased risk of skin cancer, such as:outdoor workersthose with outdoor hobbies, for example, sailing or golf.’